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Dictionaries: Disease Related Concepts  Pathways  Sponge Compounds  Mode of action 

Human Genes+Proteins ( 0 1 2 3 4 5 6 7 8 9 A B C D E F G H I J K L M N O P Q R S T U V W X Y Z
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C

CYCLIN D1 -> 
  
AKT [1]   CDK4 [1]   CDK6 [1]   CYCLIN A [1]   CYCLIN B1 [1]   INTEGRIN [1]   P-GLYCOPROTEIN [1]   P16 [1]   PI3K [1]   PROTEIN KINASE [1]   PTX-2 [1]   RAS [1]   TRANSCRIPTION FACTOR E2F [1]   WAF1 [1]   CDK2 [2]   CYCLIN E [2]   P21 [2]   P53 [2]   CYCLIN [3]   PRB [3]  
CANCER [1]   CELL PROLIFERATION [1]   CHRONIC INFLAMMATORY DISEASE [1]   CYTOTOXIC [1]   HYPERPLASIA [1]   LEUKEMIA [1]   MONOCYTIC LEUKEMIA [1]   NSCLC [1]   RHEUMATOID ARTHRITIS [1]   SENSITIVITY [1]   STEROID [1]   TUMOR CELL [1]   RETINOBLASTOMA [2]   SUPPRESSION [2]   TUMOR [2]  
G1 PHASE [1]   S PHASE [1]   SIGNAL TRANSDUCTION [1]   SIGNALING PATHWAY [1]  
ACTIVATE [1]   ACTIVATION [1]   BLOCKED [1]   CAUSED [1]   CONTROL [1]   DECREASED [1]   ENHANCED [1]   INCREASED [1]   INDUCE [1]   INDUCED [1]   PHOSPHORYLATED [1]   PRODUCTION [1]   STIMULATE [1]   SYNTHESIS [1]   TARGET [1]   ASSOCIATED [2]   BINDING [2]   DECREASE [2]   EFFECT [2]   EXPRESSION [2]   INDUCTION [2]   INHIBITED [2]   KINASE [2]   INHIBITOR [3]   PHOSPHORYLATION [3]  

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CYCLIN-DEPENDENT KINASE 2 -> 
  
ATAXIA TELANGIECTASIA MUTATED [1]   ATM [1]   BCL-2 [1]   CASPASE-3 [1]   CDK2 [1]   CYCLIN A [1]   CYCLIN E [1]   CYCLIN-DEPENDENT KINASE 5 [1]   GLYCOGEN SYNTHASE KINASE [1]   MANA [1]   MCL-1 [1]   P-GLYCOPROTEIN [1]   P-GP [1]   P53 [1]   PC-3 [1]  
CANCER [1]   CELL PROLIFERATION [1]   COLORECTAL CANCER [1]   DEATH [1]   DNA DAMAGE [1]   HEPATOCELLULAR CARCINOMA [1]   PROSTATE CANCER [1]   STRESS [1]  
ACTIVATION [1]   BINDING [1]   CAUSED [1]   COMPLEX [1]   DEPOLYMERIZATION [1]   DERIVATIVE [1]   DOWNREGULATION [1]   EXPRESSION [1]   INCREASE [1]   INDUCE [1]   INDUCED [1]   INDUCTION [1]   INHIBIT [1]   INHIBITOR [1]   INTERACTION [1]   LIGAND [1]   PHOSPHORYLATION [1]   POLYMERIZATION [1]   STIMULATED [1]   SUBSTRATE [1]   EFFECT [2]   INHIBITORY [2]   KINASE [2]   TARGET [2]  

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CYCLIN-DEPENDENT KINASE 4 -> 
  
CDK2 [1]   CDK4 [1]   P16 [1]   PRB [1]  
CANCER [1]   TUMOUR [1]  
G1/S TRANSITION [1]  
FASCAPLYSIN [1]  
BINDING [1]   CAUSED [1]   COMPLEX [1]   INACTIVATED [1]   INHIBIT [1]   INHIBITED [1]   INHIBITOR [1]   INTERFERE [1]   KINASE [1]   PHOSPHORYLATION [1]   PREVENTED [1]   PRODUCT [1]  

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CYCLIN-DEPENDENT KINASE 5 -> 
  
CYCLIN-DEPENDENT KINASE 2 [1]   GLYCOGEN SYNTHASE KINASE [1]  
BINDING [1]   COMPLEX [1]   EFFECT [1]   INHIBIT [1]   INHIBITOR [1]   INHIBITORY [1]   INTERACTION [1]   KINASE [1]   LIGAND [1]   TARGET [1]  

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CYCLIN E -> 
  
ATAXIA TELANGIECTASIA MUTATED [1]   ATM [1]   BCL-2 [1]   CASPASE-3 [1]   CDK4 [1]   CHOP [1]   CYCLIN-DEPENDENT KINASE 2 [1]   GADD153 [1]   MANA [1]   MAP KINASE [1]   MCL-1 [1]   NF-KAPPAB [1]   NRK [1]   P-GP [1]   P16 [1]   P21 [1]   PHOSPHOLIPASE C [1]   PI SYNTHASE [1]   PI4-KINASE [1]   PLC [1]   TRANSCRIPTION FACTOR E2F [1]   CYCLIN A [2]   CYCLIN D1 [2]   P-GLYCOPROTEIN [2]   P53 [2]   PC-3 [2]   CDK2 [3]   CYCLIN [3]   PRB [3]  
APOPTOSIS [1]   CANCER CELL GROWTH [1]   CELL PROLIFERATION [1]   COLORECTAL CANCER [1]   CYTOTOXIC [1]   DEATH [1]   GROWTH ARREST [1]   IMMUNODEFICIENCY [1]   LEUKEMIA [1]   MONOCYTIC LEUKEMIA [1]   NON-SMALL CELL LUNG CANCER [1]   NSCLC [1]   RETINOBLASTOMA [1]   SENSITIVITY [1]   STEROID [1]   STRESS [1]   TRANSLOCATION [1]   TUMOR [1]   TUMOR CELL [1]   DNA DAMAGE [2]   HEPATOCELLULAR CARCINOMA [2]   PROSTATE CANCER [2]   SUPPRESSION [2]   CANCER [3]  
S PHASE [2]  
AKATERPIN [1]   ILIMAQUINONE [1]  
ACTIVATE [1]   BLOCK [1]   BLOCKED [1]   DECREASED [1]   DEPOLYMERIZATION [1]   DOWNREGULATION [1]   ENHANCED [1]   INHIBIT [1]   INHIBITING [1]   METABOLITE [1]   POLYMERIZATION [1]   PRODUCE [1]   REDUCED [1]   REGULATION [1]   RIBOSYLATED [1]   SIGNAL [1]   STIMULATED [1]   SUBSTRATE [1]   SYNTHESIZED [1]   ACTIVATION [2]   ASSOCIATED [2]   BINDING [2]   CAUSED [2]   DECREASE [2]   DERIVATIVE [2]   INCREASE [2]   INDUCE [2]   INHIBITORY [2]   KINASE [2]   TARGET [2]   EFFECT [3]   INHIBITED [3]   INHIBITOR [3]   SYNTHESIS [3]   EXPRESSION [4]   INDUCED [4]   INDUCTION [4]   PHOSPHORYLATION [4]  

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CYCLOOXYGENASE-2 -> 
  
ADENYLATE CYCLASE [1]   BASIC FIBROBLAST GROWTH FACTOR [1]   BCL-2 [1]   BFGF [1]   CASPASE-3 [1]   CASPASE-9 [1]   CD31 [1]   COX-1 [1]   EP3 [1]   FAC [1]   FOLLICLE STIMULATING HORMONE RECEPTOR [1]   FSHR [1]   HO-1 [1]   HTERT [1]   IKK [1]   LHR [1]   LUTEINIZING HORMONE RECEPTOR [1]   MAC-1 [1]   MATRIX METALLOPROTEINASE-9 [1]   METALLOPROTEINASE [1]   MYELOPEROXIDASE [1]   PHOSPHODIESTERASE [1]   PHOSPHOLIPASE [1]   PLA2 [1]   PLASMINOGEN [1]   PROTEIN KINASE [1]   SPLA2 [1]   TELOMERASE REVERSE TRANSCRIPTASE [1]   THROMBOSPONDIN [1]   TNF-ALPHA [1]   TNFALPHA [1]   TSP-1 [1]   TSP-2 [1]   TUMOR NECROSIS FACTOR [1]   TYROSINE KINASE RECEPTOR [1]   UROKINASE-TYPE PLASMINOGEN ACTIVATOR [1]   VEGF-A [1]   VEGFR [1]   VEGFR-1 [1]   VEGFR-2 [1]   BAX [2]   COX [2]   P65 [2]   PGS [2]   NF-KAPPAB [3]   VEGF [3]   INOS [4]   VASCULAR ENDOTHELIAL GROWTH FACTOR [4]   COX-2 [9]  
ACUTE EXUDATIVE INFLAMMATION [1]   BONE RESORPTION [1]   BOWEL DISEASE [1]   CANCER [1]   CELL PROLIFERATION [1]   CHRONIC [1]   CHRONIC PROLIFERATIVE INFLAMMATION [1]   COLITIS [1]   CYTOTOXIC [1]   GASTRIC CANCER [1]   GRANULOMA [1]   INFILTRATION [1]   INFLAMMATION [1]   INFLAMMATORY DISEASE [1]   LEUKEMIA [1]   METASTASIS [1]   NEOVASCULARIZATION [1]   OXIDATIVE STRESS [1]   PAIN [1]   PLEURISY [1]   POUCH [1]   RHEUMATOID ARTHRITIS [1]   SOFTENING [1]   SUPPRESSION [1]   TRANSPLANTATION [1]   TUMOR [1]   TUMOR ANGIOGENESIS [1]   TUMOR CELL [1]   TUMORIGENESIS [1]   TUMOUR NECROSIS [1]   VASCULARIZATION [1]   ACUTE [2]   APOPTOSIS [2]   CHRONIC INFLAMMATION [2]   PATHOGENESIS [2]   TRANSLOCATION [3]   ANGIOGENESIS [5]  
SIGNALING PATHWAY [3]  
BOLINAQUINONE [1]   CACOSPONGIONOLIDE B [1]   PETROSASPONGIOLIDE M [1]  
ACTIVATED [1]   AFFECT [1]   ALTER [1]   ASSOCIATION [1]   BIND [1]   CAUSE [1]   CAUSED [1]   COMPLEX [1]   CONTROLLING [1]   DECREASE [1]   DOWNREGULATE [1]   DOWNREGULATION [1]   INHIBIT [1]   INHIBITING [1]   INTERFERE [1]   PHOSPHORYLATED [1]   PREVENT [1]   RECRUITED [1]   RECRUITMENT [1]   REGULATE [1]   REGULATED [1]   STIMULATED [1]   SUPPRESS [1]   SUPPRESSES [1]   SYNTHESIS [1]   UPREGULATE [1]   UPREGULATION [1]   ACTIVATOR [2]   BLOCKED [2]   ENHANCE [2]   INDUCE [2]   INHIBITORY [2]   KINASE [2]   PHOSPHORYLATION [2]   PRODUCT [2]   ASSOCIATED [3]   BINDING [3]   CONTROL [3]   EFFECT [3]   INDUCED [3]   INDUCTION [3]   REDUCTION [3]   SUPPRESSED [3]   ACTIVATION [4]   DECREASED [4]   ENHANCED [4]   INCREASED [4]   PRODUCTION [4]   REDUCED [4]   INCREASE [5]   INHIBITED [7]   INHIBITOR [7]   EXPRESSION [9]  

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CYCLOOXYGENASE 2 -> 
  
CBS [1]   COLONY-STIMULATING FACTOR [1]   COX-2 [1]   IFN-ALPHA [1]   IFN-GAMMA [1]   IGA [1]   IL-1 BETA [1]   IL-6 [1]   INOS [1]   MCP [1]   MCP-1 [1]   NF-KAPPAB [1]   RANTES [1]   RHO [1]   TNF-ALPHA [1]   TUMOR NECROSIS FACTOR [1]   TUMOR NECROSIS FACTOR ALPHA [1]   VASCULAR ENDOTHELIAL GROWTH FACTOR [1]   VEGF [1]  
ANGIOGENESIS [1]   BLIND [1]   CHRONIC [1]   CHRONIC INFLAMMATION [1]   GRANULOMA [1]  
ASSOCIATED [1]   BINDING [1]   CONTROLLING [1]   DECREASED [1]   EFFECT [1]   EXPRESSION [1]   INDUCED [1]   INHIBITED [1]   INHIBITING [1]   REDUCE [1]   REDUCED [1]   REGULATED [1]   SECRETED [1]   SECRETION [1]   ACTIVATION [2]  

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CYCLOPHILIN -> 
  
CSA [1]   CYP [1]  
ACUTE LYMPHOCYTIC LEUKEMIA [1]   COLON ADENOCARCINOMA [1]   ESCHERICHIA COLI [1]   GIANT [1]   NON-HODGKIN'S LYMPHOMA [1]   SCHISTOSOMA MANSONI [1]  
BINDING [1]   ELEVATION [1]   INCREASED [1]   METABOLISM [1]   PRODUCT [1]   STIMULATION [1]  

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CYCLOPHILIN A -> 
  
CYPA [1]   IMMUNOPHILIN [1]  
ACTIVATE [1]   ACTIVATED [1]   ACTIVATION [1]   EXPRESSION [1]   INHIBITED [1]  

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C-YES -> 
  
BKL [1]   C-FGR [1]   C-SRC [1]   FYN [1]   HCK [1]   LCK [1]   LYN [1]   MEMBRANE PROTEIN [1]   PP60C-SRC [1]   PROTEIN TYROSINE KINASE [1]   SRC [1]   SYNAPTOPHYSIN [1]   TKL [1]   TYROSINE KINASE [1]  
SIGNAL TRANSDUCTION [1]  
ELEVATED [1]   EXPRESSION [1]   KINASE [1]   PHOSPHORYLATED [1]   TRANSPORT [1]  

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